of atherosclerosis is physical activity, the mechanisms of which are not fully understood. Experimental models have

of the expression of non-coding RNAs typical for a cell type and stage of development reflect transposon activation

level has not been presented. We hypothesize that one of the key mechanisms of aging is an imbalance

in the composition of protein-coding genes in plants and animals. Therefore, the detection of expression levels

that an important trigger mechanism for it is the pathological activation of retroelements during aging, leading

, the tissue- and stage-specific activation of which plays a role in the management of the cell differentiation

to trigger FTD by activating specific retroelements. FTD is associated with multiple single nucleotide

patterns in aging. Their interaction with SARS-CoV-2 may occur at the level of RNA interference or RNA