circulating and intra-tumoral), including markers of immune response and immune
dysfunction, represent a
Beylerli, Ozal,
Beilerli, Aferin,
Ilyasova, Tatiana,
Shumadalova, Alina,
Shi, Huaizhang,
Sufianov, Albert (2024) role in AD's pathogenesis. Lastly, these circRNAs can cause mitochondrial
dysfunction, disrupting
Beylerli, Ozal,
Tamrazov, Rasim,
Gareev, Ilgiz,
Ilyasova, Tatiana,
Shumadalova, Alina,
Bai, Yunlong,
Yang, Baofeng (2023) dysfunction, and neuronal apoptosis. The ability of exosomal ncRNAs to cross the blood-brain barrier and reach
Qian, J.,
Zhuang, F.,
Chen, Y.,
Fan, X.,
Wang, J.,
Wang, Z.,
Wang, Y.,
Xu, M.,
Samorodov, A.V.,
Pavlov, V.N.,
Liang, G. (2022) significantly prevented myocardial damage and
dysfunction in T1DM mice evidenced by improved cardiac function
Nasyrova, Regina F.,
Khasanova, Aiperi K.,
Altynbekov, Kuanysh S.,
Asadullin, Azat R.,
Markina, Ekaterina A.,
Gayduk, Arseny J.,
Shipulin, German A.,
Petrova, Marina M.,
Shnayder, Natalia A. (2022) dysfunction caused by glutamate N-methyl-D-aspartate receptors (NMDARs) may represent a primary deficiency
role in atherosclerosis development has been proven, which confirms my hypothesis.
Dysfunctional foam
РОЛЬ КАРВЕДИЛОЛА В ПРЕДОТВРАЩЕНИИ АПОПТОЗА КАРДИОМИОЦИТОВ ПРИ ОСТРОМ ИНФАРКТЕ МИОКАРДАЗакирова, А.Н.,
Гарифуллин, Б.Н.,
Закирова, Н.Э.,
Zakirova, N.E.,
Garifullin, B.N.,
Zakirova, A.N. (2017) significant contribution in
myocardial remodeling and left ventricular
dysfunction after myocardial infarction