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cardiomyocytes, knockdown of Mzb1 decreased mitochondrial membrane potential, impaired mitochondrial function

because adult cardiomyocytes have already exited the cell cycle, which subseqently triggers cardiac

that regulate the cardiomyocytes differentiation of BMSCs. In this review, we chiefly summarize the regulatory

of cardiomyocyte (CM) proliferation and cardiac regeneration remains unclear. In this study, we aimed

-exposed cardiomyocytes in vitro. Overexpression of iASPP reduced the infarct size and cardiomyocyte apoptosis of mice

РОЛЬ КАРВЕДИЛОЛА В ПРЕДОТВРАЩЕНИИ АПОПТОЗА КАРДИОМИОЦИТОВ ПРИ ОСТРОМ ИНФАРКТЕ МИОКАРДАData on the mechanisms of cardiomyocyte apoptosis in myocardial infarction are presented. It has

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this regeneration ability gradually loses in the postnatal heart. Thus, the reactivation of cardiomyocyte

together, ATO blocked cardiomyocyte differentiation, induced apoptosis and cell growth arrest during

Rutin contents in biological samples of blood plasma, cardiomyocytes, and mitochondria

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