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ФЕНОМЕН РОЛИ КОЛЛАГЕНА IV ТИПА И МАТРИКСНОЙ МЕТАЛЛОПРОТЕИНАЗЫ 9 ТИПА В РЕМОДЕЛИРОВАНИИ МИОКАРДА ЛЕВОГО ЖЕЛУДОЧКА ПРИ ИШЕМИЧЕСКОЙ БОЛЕЗНИ СЕРДЦА
ROLE OF TYPE IV COLLAGEN AND MATRIX METALLOPROTEINASE-9 IN REMODELING OF THE LEFT VENTRICULAR IN CORONARY ARTERY DISEASE
ПОПОВ, М.А., ШУМАКОВ, Д.В., ЗЫБИН, Д.И., ГУРЕВИЧ, Л.Е., АШЕВСКАЯ, В.Е., БАБОКИН, В.Е., ПРОНИНА, В.П., Popov M.A., Shumakov D.V., Zybin D.I., Gurevich L.E., Ashevskaya V.E., Babokin V.E., Pronina V.P. (2019)
BASAL MEMBRANE OF CARDIOMYOCYTES
Mzb1 protects against myocardial infarction injury in mice via modulating mitochondrial function and alleviating inflammation
Zhang, Lu, Wang, Yi-ning, Ju, Jia-ming, Shabanova, Azaliia, Li, Yue, Fang, Ruo-nan, Sun, Jia-bin, Guo, Ying-ying, Jin, Tong-zhu, Liu, Yan-yan, Li, Tian-yu, Shan, Hong-li, Liang, Hai-hai, Yang, Bao-feng (2020)
cardiomyocytes, knockdown of Mzb1 decreased mitochondrial membrane potential, impaired mitochondrial function
Regulation of cardiomyocyte fate plasticity: a key strategy for cardiac regeneration
Gong, R., Jiang, Z., Zagidullin, N., Liu, T., Cai, B. (2021)
because adult cardiomyocytes have already exited the cell cycle, which subseqently triggers cardiac
CARDIOMYOCYTE DIFFERENTIATION OF MESENCHYMAL STEM CELLS FROM BONE MARROW: NEW REGULATORS AND ITS IMPLICATIONS
GUO, X., BAI, Y., ZHANG, L., ZHANG, B., DU, Z., CAI, B., ZAGIDULLIN, N., CARVALHO, K.3 (2018)
that regulate the cardiomyocytes differentiation of BMSCs. In this review, we chiefly summarize the regulatory
CARDIOMYOCYTE DIFFERENTIATION OF MESENCHYMAL STEM CELLS FROM BONE MARROW: NEW REGULATORS AND ITS IMPLICATIONS
Guo, X., Bai, Y., Zhang, L., Zhang, B., Zagidullin, N., Carvalho, K., Du, Z., Cai, B. (2018)
that regulate the cardiomyocytes differentiation of BMSCs. In this review, we chiefly summarize the regulatory
ALKBH5 regulates cardiomyocyte proliferation and heart regeneration by demethylating the mRNA of YTHDF1
Han, Z.B., Wang, X.X., Xu, Z.H., Cao, Y., Gong, R, Zagidullin, N., Sukhareva, N., Zhang, Y. (2021)
of cardiomyocyte (CM) proliferation and cardiac regeneration remains unclear. In this study, we aimed
Corrigendum to: iASPP protects the heart from ischemia injury by inhibiting p53 expression and cardiomyocyte apoptosis
Yagudin, T., Zhao, Y., Gao, H., Zhang, Y., Yang, Y., Zhang, X., Ma, W., Daba, T.M., Ishmetov, V., Kang, K., Yang, B., Pan, Z. (2020)
-exposed cardiomyocytes in vitro. Overexpression of iASPP reduced the infarct size and cardiomyocyte apoptosis of mice
iASPP protects the heart from ischemia injury by inhibiting p53 expression and cardiomyocyte apoptosis
Yagudin, T, Zhao, Y, Gao, HY, Zhang, Y, Yang, Y, Zhang, XF, Ma, WB, Daba, TM, Ishmetov, V, Kang, K, Yang, BF, Pan, ZW (2021)
-exposed cardiomyocytes in vitro. Overexpression of iASPP reduced the infarct size and cardiomyocyte apoptosis of mice
Cyclin L1 controls cardiomyocyte proliferation and heart repair after injury
Gong, Rui, Gao, Xinlu, Liu, Yu, Shen, Yifu, Jiang, Zuke, Wang, Xiuxiu, Zagidullin, Naufal, Ma, Wenya, Wang, Ning, Cai, Benzhi (2023)
Cyclin L1 controls cardiomyocyte proliferation and heart repair after injury
РОЛЬ КАРВЕДИЛОЛА В ПРЕДОТВРАЩЕНИИ АПОПТОЗА КАРДИОМИОЦИТОВ ПРИ ОСТРОМ ИНФАРКТЕ МИОКАРДА
THE ROLE OF CARVEDILOL IN THE PREVENTION OF CARDIOMYOCYTE APOPTOSIS CAUSED BY ACUTE MYOCARDIAL INFARCTION
Закирова, А.Н., Гарифуллин, Б.Н., Закирова, Н.Э., Zakirova, N.E., Garifullin, B.N., Zakirova, A.N. (2017)
Data on the mechanisms of cardiomyocyte apoptosis in myocardial infarction are presented. It has

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