Erdman, V.V.,
Karimov, D.D.,
Tuktarova, I.A.,
Petintseva, A.A.,
Timasheva, Y.R.,
Nasibullin, T.R. (2025) of the SEMA6A (Yb8NBC597*ID, HR = 0.54, P = 0.016 for the cerebrovascular
diseases), TEAD1 (Ya5ac2013*II, HR
in the scientific literature, the presence of 467 transposon-derived miRNA
genes in the
human genome has been
properties and exert inhibitory effects on
human transposable elements. Viruses and aging have been suggested
role in Alzheimer's
disease development. Still, they cannot explain all cases of the
disease and cannot
Abstract: The dysregulation of transposable elements plays a key role in
human carcinogenesis
Влияние транспозонов на развитие болезни АльцгеймераInvolvement of transposable elements in Alzheimer’s
disease pathogenesis
of individuals belonging to different
human populations. The possible insertion of SARS-CoV-2 c
changes in atherosclerosis pathogenesis. The
disease affects the cardiovascular system in the adult