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of the SEMA6A (Yb8NBC597*ID, HR = 0.54, P = 0.016 for the cerebrovascular diseases), TEAD1 (Ya5ac2013*II, HR

in the scientific literature, the presence of 467 transposon-derived miRNA genes in the human genome has been

properties and exert inhibitory effects on human transposable elements. Viruses and aging have been suggested

role in Alzheimer's disease development. Still, they cannot explain all cases of the disease and cannot

Abstract: The dysregulation of transposable elements plays a key role in human carcinogenesis

АНАЛИЗ АССОЦИАЦИЙ ПОЛИМОРФНОГО ЛОКУСА 32454C>T ГЕНА JAK1 C ДОЛГОЛЕТИЕМhuman longevity

Влияние транспозонов на развитие болезни АльцгеймераInvolvement of transposable elements in Alzheimer’s disease pathogenesis

of individuals belonging to different human populations. The possible insertion of SARS-CoV-2 c

ЭПИГЕНЕТИЧЕСКИЕ МЕХАНИЗМЫ ВЗАИМОСВЯЗИ ОСТЕОАРТРИТА СО СТАРЕНИЕМ

changes in atherosclerosis pathogenesis. The disease affects the cardiovascular system in the adult

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