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of cardiomyocyte (CM) proliferation and cardiac regeneration remains unclear. In this study, we aimed

of targeting cellular fate plasticity to induce cardiomyocyte proliferation or formation, and also provide

Cyclin L1 controls cardiomyocyte proliferation and heart repair after injury

significant inhibition of proliferation during the whole process of cardiac differentiationofhi

DACH1 silencing-induced cardiomyocyte proliferation. In addition, targeting a conserved fragment of Lnc

that regulate the cardiomyocytes differentiation of BMSCs. In this review, we chiefly summarize the regulatory

CARDIOMYOCYTE DIFFERENTIATION OF MESENCHYMAL STEM CELLS FROM BONE MARROW: NEW REGULATORS AND ITS IMPLICATIONS that regulate the cardiomyocytes differentiation of BMSCs. In this review, we chiefly summarize the regulatory

-exposed cardiomyocytes in vitro. Overexpression of iASPP reduced the infarct size and cardiomyocyte apoptosis of mice

-exposed cardiomyocytes in vitro. Overexpression of iASPP reduced the infarct size and cardiomyocyte apoptosis of mice

Myocardial infarction (MI) leads to the loss of cardiomyocytes, left ventricle dilation and cardiac

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