Функциональный дуализм транскриптов транспозонов в эволюции геномов эукариот processes into regulatory networks contributed, simultaneously with the
complication of eukaryotes
Ye, L.,
Chen, X.,
Wang, M.,
Jin, L.,
Zhuang, Z.,
Yang, D.,
Guan, X.,
Samorodov, A.V.,
Pavlov, V.N.,
Chattipakorn, N.,
Feng, J.,
Wang, Y.,
Luo, W.,
Liang, G. (2021) developed an anti-inflammatory compound C66, which prevented inflammatory diabetic
complications via
Onorato, EM,
Alamanni, F,
Muratori, M,
Smolka, G,
Wojakowski, W,
Pysz, P,
Zorinas, A,
Zakarkaite, D,
Eltchaninoff, H,
Litzer, PY,
Godart, F,
Calvert, P,
Christou, C,
Mussayev, A,
Missiroli, B,
Buzaev, I,
Curello, S,
Tesorio, T,
Bartorelli, AL (2022) (three patients), residual leak (two patients) and vascular
complication (one patient). During follow
as possible diagnostic and
treatment targets. Here, we discuss how claudin-related signaling pathways affect
Zelinska, N.,
Iotova, V.,
Skorodok, J.,
Malievsky, O.,
Peterkova, V.,
Samsonova, L.,
Rosenfeld, Ron G.,
Zadik, Zvi,
Jaron-Mendelson, Michal,
Koren, Ronit,
Amitzi, Leanne,
Raduk, Dmitri,
Hershkovitz, Oren,
Hart, Gili (2017) of
treatment with either
MOD-4023 (N = 42) or r-hGH (N = 11).
Interventions: C-terminal peptide–modified h