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-coding RNAs (lncRNAs) acting as competing endogenous RNAs (ceRNAs) continues to grow, encompassing a broad

, regulatory mechanisms, and therapeutic potential of ncRNAs including microRNA (miRNA), long ncRNA (lnc

-coding RNAs (lncRNAs), involve in the progress of multiple cardiovascular diseases. However, their potential

) and long non-coding RNAs (lncRNAs), as key regulators in the pathogenesis of hypertension. These ncRNAs

, and MEG3 lncRNAs in patients with type 2 diabetes and metabolic parameters, as well as the risk

(ncRNAs), specifically microRNAs (miRNAs) and long non-coding RNAs (lncRNAs), present promising

of long noncoding RNAs and microRNAs from transposons, changes in the expression of which are observed

regulators, such as long noncoding RNAs (lncRNAs). To assess the contribution of genes involved in key

regulators, such as long noncoding RNAs (lncRNAs). To assess the contribution of genes involved in key

function, and acceleration of cellular senescence. Long noncoding RNAs (lncRNAs) function as critical

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