Gilyazova, Irina,
Gimalova, Galiya,
Nizamova, Aigul,
Nizamova, Aigul,
Ishbulatova, Ekaterina,
Pavlov, Valentin,
Khusnutdinova, Elza (2024) , regulatory mechanisms, and therapeutic potential of ncRNAs including micro
RNA (mi
RNA), long nc
RNA (
lncRNAJiang, Jianhao,
Gareev, Ilgiz,
Ilyasova, Tatiana,
Shumadalova, Alina,
Du, Weijie,
Yang, Baofeng (2024) -established ce
RNA networks, involving classical interactions between
lncRNAs, microRNAs (miRNAs), and messenger
and Alzheimer's disease originated from SINE/MIRs that evolved from tRNAs, the role of tRNAs and the tRFs and t
RNAYuan, Yue,
Han, Xuejie,
Zhao, Xinbo,
Zhang, Haiyu,
Vinograd, Asiia,
Bi, Xin,
Duan, Xiaoxu,
Cao, Yukai,
Gao, Qiang,
Song, Jia,
Sheng, Li,
Li, Yue (2024) , these circulating exosome
lncRNA levels were different between paroxysmal and persistent AF patients, which were
, and MEG3
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Korytina, G.F.,
Akhmadishina, L.Z.,
Markelov, V.A.,
Nasibullin, T.R.,
Aznabaeva, Y.G.,
Kochetova, O.V.,
Khusnutdinova, N.N.,
Larkina, A.P.,
Zagidullin, N. Sh.,
Victorova, T.V. (2024) function, and acceleration of cellular senescence. Long noncoding RNAs (
lncRNAs) function as critical
Markelov, V.A.,
Korytina, G.F.,
Aznabaeva, Y.G.,
Gibadullin, I.A.,
Akhmadishina, L.Z.,
Nasibullin, T.R.,
Kochetova, O.V.,
Avzaletdinov, A.M.,
Zagidullin, N. Sh. (2024) of
lncRNA (TP53TG1, LINC00342, H19, MALAT1, DNM3OS, and MEG3) and protein-coding (PTEN, TGFB2, FOXO3
Markelov, V.A.,
Korytina, G.F,
Aznabaeva, Y.G.,
Gibadullin, I.A.,
Akhmadishina, L.Z.,
Nasibullin, T.R.,
Kochetova, O.V.,
Avzaletdinov, A.M.,
Zagidullin, N Sh (2024) of
lncRNA (TP53TG1, LINC00342, H19, MALAT1, DNM3OS, and MEG3) and protein-coding (PTEN, TGFB2, FOXO3
Beylerli, Ozal,
Shi, Huaizhang,
Begliarzade, Sema,
Shumadalova, Alina,
Ilyasova, Tatiana,
Sufianov, Albert (2024) (ncRNAs), specifically microRNAs (miRNAs) and long non-coding RNAs (
lncRNAs), present promising
RNAs) and long non-coding RNAs (
lncRNAs), as key regulators in the pathogenesis of hypertension. These nc