Bai, Ming,
Wang, Xiaolong,
Zhang, Huixue,
Wang, Jianjian,
Lyaysan, Gaysina,
Xu, Si,
Tian, Kuo,
Wang, Tianfeng,
Li, Jie,
Wang, Na,
Lu, Xiaoyu,
Zhang, Xiaoming,
Wang, Lihua (2022) investigated
its prognostic value in GBM and LGG, respectively. Focusing on the subclonal mutations, our
Lin, Jianjun,
Lin, Ke,
Huang, Lijiang,
Jiang, Yongsheng,
Ding, Xiaoxiao,
Luo, Wu,
Samorodov, Aleksandr V.,
Pavlov, Valentin N.,
Liang, Guang,
Qian, Jianchang,
Wang, Yi (2022) with TLR4 and
its adaptor protein myeloid differentiation protein 2 (MD2). Genetic loss of MD2 ameliorates
Vaiman, E.E.,
Shnayder, N.A.,
Zhuravlev, N.M.,
Petrova, M.M.,
Asadullin, A.R.,
Al-Zamil, M.,
Garganeeva, N.R.,
Shipulin, G.A.,
Cumming, G.,
Nasyrova, R.F. (2022) on AP-induced LQTS and proposes a
new personalized approach to assessing the risk of
its development
Lu, Enzhou,
Gareev, Ilgiz,
Yuan, Chao,
Liang, Yanchao,
Sun, Jingxian,
Chen, Xin,
Beylerli, Ozal,
Sufianov, Albert,
Zhao, Shiguang,
Yang, Guang (2022) damage, and blockage of related apoptosis pathways play an important role in
it. It is known
of neuropharmacological interaction.
Materials and methods: To study the mechanism of action of N-199/1,
its effect on 5
Wei, XM,
Zhang, F,
Qiu, YK,
Shen, H,
Ilyasova, T,
Liu, L. (2022) Objective
It is well known that accurate location of the leak in the operation is crucial