Zhu, Weiwei,
Luo, Wu,
Han, Jibo,
Zhang, Qiuyan,
Ji, Lijun,
Samorodov, Aleksandr V.,
Pavlov, Valentin N.,
Zhuang, Zaishou,
Yang, Daona,
Yin, Lina,
Huang, Lijiang,
Liang, Guang (2023) identified MyD88 as a direct target of Sch B for its
anti-inflammatory activity, and suggest that Sch B may
Li, Xiang,
Yin, Lina,
Liao, Jing,
Yang, Jun,
Cai, Binhao,
Yu, Yiming,
Su, Sijia,
Du, Zhiteng,
Li, Xiaobo,
Zhou, Ying,
Chen, Pan,
Cho, Won-Jea,
Chattipakorn, Nipon,
Samorodov, Aleksandr V.,
Pavlov, Valentin N.,
Zhang, Fengzhi,
Liang, Guang,
Tang, Qidong (2023) and synthesized using cinnamic acid as the lead compound. We tested the preliminary
anti-inflammatory activity in the
inflammatory response, chemokine
activity, and immune response. The KEGG analysis identified several pathways
Agaverdiev, Murad,
Shamsov, Bedil,
Mirzoev, Sorbon,
Vardikyan, Andranik,
Encarnacion, Manuel Ramirez,
Nurmukhametov, Renat,
Beilerli, Aferin,
Zhang, Bohan,
Gareev, Ilgiz,
Pavlov, Valentin (2023) of the function of the cellular component of SVF as an
anti-inflammatory, pro-angiogenic and cell differentiation
. MSCs within SVF release
anti-inflammatory cytokines such as interleukin-10 (IL-10) and transforming
Lin, Jianjun,
Lin, Ke,
Huang, Lijiang,
Jiang, Yongsheng,
Ding, Xiaoxiao,
Luo, Wu,
Samorodov, Aleksandr V.,
Pavlov, Valentin N.,
Liang, Guang,
Qian, Jianchang,
Wang, Yi (2022) heme-induced inflammation and
inflammatory cytokine production in the spleen of MD2 knockout (MD2
Gilyazova, Irina,
Asadullina, Dilara,
Kagirova, Evelina,
Sikka, Ruhi,
Mustafin, Artur,
Ivanova, Elizaveta,
Bakhtiyarova, Ksenia,
Gilyazova, Gulshat,
Gupta, Saurabh,
Khusnutdinova, Elza,
Gupta, Himanshu,
Pavlov, Valentin (2023) inflammatory processes, haemopoiesis, allergic responses, and other key aspects of the innate immune system
Larionova, Irina,
Iamshchikov, Pavel,
Kazakova, Anna,
Rakina, Militsa,
Menyalo, Maxim,
Enikeeva, Kadriia,
Rafikova, Guzel,
Sharifyanova, Yuliya,
Pavlov, Valentin,
Villert, Alisa,
Kolomiets, Larisa,
Kzhyshkowska, Julia (2025) that chemotherapy inhibited interferon-dependent signaling pathways, but
activated some TGFb-related genes. Our