Изменения иммунной системы в патогенезе нейрофиброматоза 1-го типа into the tumor microenvironment, which
actively degranulate. The released cytokines promote neoangiogenesis
of ketotiphen, Lydase and Aevit, as well as monotherapy with an ATP-independent inhibitor of mitogen-
activated activity of histone acetylases in the IL-6 promoter area. In contrast, the expression of anti
in the programmed
activation of mobile genetic elements, which is reflected in changes in the body’s epigenetic
, the only effective drugs for the treatment of tumor syndrome in NF1 are inhibitors of mitogen-
activated