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NEW BIOMARKERS OF CARDIOVASCULAR ENDPOINTS AND HEART FAILURE PROGRESSION ANALYSIS AFTER MYOCARDIAL INFARCTION
ZAGIDULLIN, N.SH., HAMITOVA, A., LACKMAN, I., IONIN, V. (2019)
Myocardial infarction (MI) is one of the main factors in heart failure (HF) progression. New
Antithrombotic Activity of the Antiplatelet Agent Angipur on the Model of Arterial Thrombosis in Rats with Isoproterenol-Induced Myocardial Infarction
Spasov, AA, Kucheryavenko, AF, Khaliullin, FA, Gurova, NA, Sirotenko, VS, Samorodov, AV, Gaidukova, KA, Pavlov, VN (2022)
comorbidities and with isoproterenol-induced myocardial infarction. In animals without comorbidities, Angipur
ПУТИ ОПТИМИЗАЦИИ МОРФОЛОГИЧЕСКОЙ ДИАГНОСТИКИ РАЗЛИЧНЫХ ТИПОВ ИНФАРКТА МИОКАРДА
OPTIMIZATION WAYS TO THE MORPHOLOGICAL DIAGNOSIS OF VARIOUS MYOCARDIAL INFARCTION TYPES
МУСТАФИН, ТАГИР ИСЛАМНУРОВИЧ, ЩЕКИН, СЕРГЕЙ ВИТАЛЬЕВИЧ, ЩЕКИН, ВЛАС СЕРГЕЕВИЧ, MUSTAFIN T.I., SHCHEKIN S.V., SHCHEKIN V.S. (2020)
OPTIMIZATION WAYS TO THE MORPHOLOGICAL DIAGNOSIS OF VARIOUS MYOCARDIAL INFARCTION TYPES
TCTAP C-025 EMBOLIC MYOCARDIAL INFARCTION CAUSED BY LEFT ATRIUM EMBOLI FROM ATRIAL FIBRILLATION
Zagitov, I.G., Plechev, V.V., Nikolaeva, I.E., Buzaev, I.V. (2016)
TCTAP C-025 EMBOLIC MYOCARDIAL INFARCTION CAUSED BY LEFT ATRIUM EMBOLI FROM ATRIAL FIBRILLATION
МОРФОЛОГИЧЕСКИЕ И ЛАБОРАТОРНО-ФУНКЦИОНАЛЬНЫЕ ПАРАЛЛЕЛИ ПРИ ДИАГНОСТИКЕ ИНФАРКТА МИОКАРДА 2-ГО ТИПА
MORPHOLOGICAL AND LABORATORY-FUNCTIONAL PARALLELS IN DIAGNOSTICS OF MYOCARDIAL INFARCTION TYPE II
Т. И. Мустафин, T. I. Mustafin, С. В. Щекин, S. V. Shchekin, В. С. Щекин, V. S. Shchekin (Медицинский вестник Башкортостана, №2, 2021)
MORPHOLOGICAL AND LABORATORY-FUNCTIONAL PARALLELS IN DIAGNOSTICS OF MYOCARDIAL INFARCTION TYPE II
Corrigendum to: iASPP protects the heart from ischemia injury by inhibiting p53 expression and cardiomyocyte apoptosis
Yagudin, T., Zhao, Y., Gao, H., Zhang, Y., Yang, Y., Zhang, X., Ma, W., Daba, T.M., Ishmetov, V., Kang, K., Yang, B., Pan, Z. (2020)
of this study was to investigate the possible role of iASPP in acute myocardial infarction. The protein level
iASPP protects the heart from ischemia injury by inhibiting p53 expression and cardiomyocyte apoptosis
Yagudin, T, Zhao, Y, Gao, HY, Zhang, Y, Yang, Y, Zhang, XF, Ma, WB, Daba, TM, Ishmetov, V, Kang, K, Yang, BF, Pan, ZW (2021)
of this study was to investigate the possible role of iASPP in acute myocardial infarction. The protein level
ОСОБЕННОСТИ РЕМОДЕЛИРОВАНИЯ ЛЕВОГО ЖЕЛУДОЧКА У БОЛЬНЫХ ОСТРЫМ ИНФАРКТОМ МИОКАРДА С ПОДЪЕМОМ И БЕЗ ПОДЪЕМА СЕГМЕНТА ST
LEFT VENTRICULAR REMODELING IN PATIENTS WITH ACUTE MYOCARDIAL INFARCTION WITH AND WITHOUT ST SEGMENT ELEVATION
Закирова, Н.Э., Казиева, З.А., Закирова, А.Н., Zakirova, N.E., Kazieva, Z.A., Zakirova, A.N. (2016)
(STEMI) and non-ST segment elevation (non-STEMI) myocardial infarction (MI). Materials and methods
ПРОГНОСТИЧЕСКИЕ МАРКЕРЫ ТЯЖЕСТИ ПОСТИНФАРКТНОГО ТЕЧЕНИЯ У ПАЦИЕНТОВ ТРУДОСПОСОБНОГО ВОЗРАСТА, ПЕРЕНЕСШИХ Q-ИНФАРКТ МИОКАРДА
PREDICTIVE MARKERS OF SEVERITY OFPOSTINFARCTION IN PATIENTS OF WORKING AGE WHO HAVE HAD Q-MYOCARDIAL INFARCTION
БАБУШКИНА, Г.В., ПЕРМЯКОВА, С.В., ГУБАЕВА, А.М., BABUSHKINA, G.V., PERMYAKOVA, S.V., GUBAEVA, A.M. (2020)
PREDICTIVE MARKERS OF SEVERITY OFPOSTINFARCTION IN PATIENTS OF WORKING AGE WHO HAVE HAD Q-MYOCARDIAL
РОЛЬ КАРВЕДИЛОЛА В ПРЕДОТВРАЩЕНИИ АПОПТОЗА КАРДИОМИОЦИТОВ ПРИ ОСТРОМ ИНФАРКТЕ МИОКАРДА
THE ROLE OF CARVEDILOL IN THE PREVENTION OF CARDIOMYOCYTE APOPTOSIS CAUSED BY ACUTE MYOCARDIAL INFARCTION
Закирова, А.Н., Гарифуллин, Б.Н., Закирова, Н.Э., Zakirova, N.E., Garifullin, B.N., Zakirova, A.N. (2017)
Data on the mechanisms of cardiomyocyte apoptosis in myocardial infarction are presented. It has

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