Galimova, E.F.,
Gromenko, I.D.,
Gilyazova, I.R.,
Gromenko, D.D.,
Galimov, K.Sh.,
Abdeeva, G.R.,
Gromenko, Yu.Yu.,
Khusnutdinova, E.K.,
Galimov, Sh.N. (2024) . The
expression of exosomal miR-449c and miR-135a was significantly different in the experimental group (
p = 0
Xu, Dongxiao,
Gareev, Ilgiz,
Beylerli, Ozal,
Pavlov, Valentin,
Le, Huang,
Shi, Huaizhang (2024) with the formation and progression of IAs. Methods: The investigation gathered
expression data of miRNAs (from GSE
supply to tissues and organs. These processes are characterized by changes in the
expression of the CACNA
УРОВЕНЬ ЭКСПРЕССИИ CART-ПЕПТИДА В ЯДРАХ АМИГДАЛЫ МОЗГА КРЫСХИСМАТУЛЛИНА, З.Р.,
САДРТДИНОВА, И.И.,
ФЕДОРОВА, А.М.,
ФАЙРУШИНА, А.И.,
ДАВЛЕТБАЕВА, А.Р.,
Khismatullina, Z.R.,
Sadrtdinova, I.I.,
Fedorova, A.M.,
Fairushina, A.I.,
Davletbaeva, A.R. (2024) -KO исключительно в центральном ядре - Се (
p≤0,05), что доказывает определение, что центральное ядро МТ является
Asadullina, D.D.,
Gilyazova, I.R.,
Ivanova, E.A.,
Izmailova, S.M.,
Gilyazova, G.R.,
Pavlov, V.N.,
Khusnutdinova, E.K. (2024) , -503, -144)
expression levels before and after ICI therapy in plasma samples obtained from 42 cc
Gareev, Ilgiz,
Beylerli, Ozal,
Ahmad, Aamir,
Ilyasova, Tatiana,
Shi, Huaizhang,
Chekhonin, Vladimir (2024) . Today, it is known that the disruption of gene
expression is one of the main mechanisms that contribute