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СПОСОБ МОДЕЛИРОВАНИЯ РЕЦЕССИИ ДЕСНЫ

. The animals were divided into 3 groups. The first group consisted of organs of 10 mice that received a dose

ИНДУКЦИЯ ИММУННОГО ОТВЕТА, ВЫЗВАННАЯ РАСТВОРОМ G 01, У ЛАБОРАТОРНЫХ ЖИВОТНЫХ С ПЕРЕВИТОЙ МЕЛАНОМОЙ (В 16)INDUCTION OF IMMUNE RESPONSE CAUSED BY G-01 SOLUTION IN LABORATORY ANIMALS WITH TRANSPLANTED

into the present, “comes to life” in it and is transformed into the future. In humans, unlike in animals, signs

ПРОТИВООПУХОЛЕВАЯ АКТИВНОСТЬ РАСТВОРА ПРИРОДНОГО ВЕЩЕСТВА GK01 НА ПРИМЕРЕ САРКОМЫ МK1 БЕЗ НАЛИЧИЯ АДЪЮВАНТА

procedures were approved by the Institutional Animal Care and Use Committee of Harbin Medical University

to the control group. The histological examination of the myocardium of the animals treated with PVSHE showed

in the composition of protein-coding genes in plants and animals. Therefore, the detection of expression levels

-inflammatory mechanism of Sch B in macrophages and in an animal model of ALI. Methods: qPCR array was used to probe

Противоопухолевая активность комбинации дегидроксиметилэпоксихиномицина (DHMEQ) и цисплатина на модели диссеминированного рака яичниковanimal model of disease

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