LIU, Z.,
ZHANG, R.,
CHEN, X.,
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LIU, W.,
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SOKHATSKII, A.,
GAREEV, I. (2019) for differentially expressed
genes (DEGs) in ICH, with
a fold change (FC) value of (|log2FC|) > 2 and
a P-value of <0
loci in CCL20 (rs6749704) and CCL5 (rs2107538)
genes with type 2 diabetes had been identified as
aFEDOROVA, Y.Y.,
KARUNAS, A.S.,
GIMALOVA, G.F.,
KHUSNUTDINOVA, E.K.,
SAVELIEVA, O.N,
MURZINA, R.R.,
GATIYATULLIN, R.F.,
ETKINA, E.I. (2019) established that, in Tatars, the rs37973 G allele of the GLCCI1
gene is
a marker of an increased risk
WANG, K.,
LIU, H.,
LIU, J.,
WANG, X.,
TENG, L.,
ZHANG, J.,
CHEN, X.,
WANG, N.,
ZHONG, Y.,
HOU, X.,
JIANG, H.,
ZHANG, X.,
ZHAO, S.,
LIU, Y.,
YAO, Y.,
WANG, J.,
QU, Y.,
PENG, F.,
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LIAO, X. (2019) confirmed that IL1RN is
a tumor associated
gene and its expression is negatively correlated
KORYTINA, G.F.,
AKHMADISHINA, L.Z.,
KOCHETOVA, O.V.,
VIKTOROVA, T.V.,
AZNABAEVA, Y.G.,
ZAGIDULLIN, N.SH.,
ZAGIDULLIN, SH.Z.,
KZHYSHKOWSKA, J.G (2019) Background and objective: Chronic obstructive pulmonary disease (COPD) is
a complex chronic
PSHENNIKOVA, V.G.,
BARASHKOV, N.A.,
ROMANOV, G.P.,
TERYUTIN, F.M.,
SOLOV'EV, A.V.,
GOTOVTSEV, N.N.,
NIKANOROVA, A.A.,
FEDOROVA, S.A.,
NAKHODKIN, S.S.,
SAZONOV, N.N.,
MOROZOV, I.V.,
BONDAR, A.A.,
POSUKH, O.L,
DZHEMILEVA, L.U.,
KHUSNUTDINOVA, E.K. (2019) predictions for missense variants of the GJB2 (Cx26), GJB
6 (Cx30), and GJB3 (Cx31) connexin
genes associated
Paar, V.,
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Brandt, M.C.,
Lichtenauer, M.,
Hoppe, U.C.,
Motloch, L.J. (2019) . We highlight the therapeutic potential of
gene therapy as
a novel and innovative approach for future
ASSOCIATIONS OF THE NRF2/KEAP1 PATHWAY AND ANTIOXIDANT DEFENSE GENE POLYMORPHISMS WITH CHRONIC OBSTRUCTIVE PULMONARY DISEASEKorytina, G.F.,
Akhmadishina, L.Z.,
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Viktorova, T.V. (2019) Background and objective: Chronic obstructive pulmonary disease (COPD) is
a complex chronic
Objective - The aim of the work was to study the contribution of the immune response mediator
genesНАСЛЕДСТВЕННЫЙ РАК ЯИЧНИКОВ: ВКЛАД ИЗМЕНЕНИЙ ГЕНОВ-КАНДИДАТОВ В ПАТОГЕНЕЗ ЗАБОЛЕВАНИЯФаисханова, Р.Р.,
Прокофьева, Д.С.,
Хуснутдинова, Э.К.,
Сакаева, Д.Д.,
Гордиев, М.Г.,
Faiskhanova, R.R.,
Prokofieva, D.S.,
Khusnutdinova, E.K.,
Sakaeva, D.D.,
Gordiev, M.G. (2019) . Приблизительно
6% наследственного РЯ приходится на герминальные мутации в генах BARD1, BRIP1, CHEK, MRE11
A, MSH
6